untitled-1STEROIDS WITH THE HIGHEST ANDROGENIC INDEX

1) methyltrienolone (6000)
2) fluoxymesterone (800)
3) trenbolone (500)

STEROIDS WITH THE LOWEST ANDROGENIC INDEX

1) oxandrolone (24)
2) stanozolol (30)
3) nandrolone (37)
4) oxymetholone (45)
5) boldenone (50)
6) methenolone (57)

SLOW RELEASE INJECTABLE AAS

1) testosterone enanthate
2) testosterone cypionate
3) testosterone undecaonate (per os)
4)  testosterone decaonate
5) methenolone enanthate (DHT derivative)
6) trenbolone enanthate (19nor derivative)
7) nandrolone undecaonate (19nor derivative)
8) boldenone undecyclate (testosterone derivative)

IMMEDIATE RELEASE INJECTABLE AAS

1) stanozolol suspension (DHT derivative)
2) nandrolone phenylpropionate (19nor derivative)
3) testosterone propionate
4) testosterone suspension
5) drostenolone propionate (DHT synthetic)
6) trenbolone acetate (19nor derivative)
7) trenbolone suspension-base (19nor derivative)

AROMATIZED AAS

1) testosterone
2) oxymetholone? (DHT derivative)
3) methyldrostenolone (methyltestosterone derivative)
4) methyltestosterone (testosterone derivative)
5) nandrolone undecaonate (19nor derivative)

NON AROMATIZED AAS (DHT derivatives)

1) stanozolol
2) oxandrolone
3) mesterolone
4) methenolone
5) fluoxymesterone (testosterone derivative)
6) trenbolone acetate (19nor derivative)
7) drostenolone propionate (DHT synthetic)

The higher androgenic index a steroid has, the more effective is for beta oxidation-lipolysis of the subcutaneous tissue, leading to muscle hardness.

On the other hand, there is a higher reduction rate to dihydrotestosterone, leading to andogenic side effects such as benign prostatic hyperplasia, male pattern baldness-androgenic alopecia, acne, oily skin, body/facial hair growth, aggressive behavior, as well as erythrocytosis.

Anabolic steroids having a lower androgenic index than testosterone have the ability to cause a positive nitrogen balance-tissue anabolism and act against catabolism.

Furthermore, the androgenic side effects (acne, hirsutism, neuropsychiatric disorders, prostate enlargement, male-pattern baldness, suppression of the hypothalamic pituitary testicular axis, neuropsychiatric disorders) will be less, but always are dose and time dependent.

PHARMAKOKINETICS OF AAS

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